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flux and the cleaning of the common biliary duct (CBD) may prevent potentially avoidable recurrent pancreatitis.
METHODS: In the period September 1997/December 2008 we have treated 224 ABP (34 severe, 190 mild/moderate):
162 (72,4%) with the first attack, 62 (27,6%) with recurrent ABP (second or further attack). The patients with recurrent
pancreatitis had not undergone, in the previous hospital stay elsewhere, the evaluation and, if necessary, the treatment
of the papillary obstacle and /or CBD stones, sludge, etc. In our hospital all patients had undergone the treatment
of ABP. The treatment was completed with cholecystectomy. All the patients, after the discharge, were introduced in a
follow-up program (clinical and ultrasonoghaphic (US) control after 180 days and 1 year).
RESULTS: In the follow-up of recurrent pancreatitis we have controlled 35 patients (56%-27 lost). The results of the follow-
up showed, beside the absence of recurrent acute episodes, the stable normalization of laboratory cholestasis tests and
US control. The same controls in 78 patients ( 48,1%) with a first attack of acute pancreatitis resulted normal in
absence of a new acute episode.
CONCLUSIONS: Recurrent ABP have been caused by persistent papillary obstacle. Therefore we confirm therapeutic validity
of the instrumental control (US/MRCP) and the possible treatment of papillary or biliary lithiasic obstacle for the
prevention of recurrent ABP.